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Genetic mutation

A genetic mutation is any stable and heritable change in the sequence of DNA nucleotides. The agents that cause them (mutagens) can be chemical (eg nitrous acid), physical (ultraviolet radiation between others) or biological (such as viruses). In these cases, these changes could lead to different genetic diseases.

There are three types of genetic mutations: molecular mutations, chromosomal mutations and genomic mutations

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Recent Posts

  • NCBI References of Lab polyclonals
  • Suppliers of Lab recombinants
  • Distributors of Lab monoclonals
  • Compare monoclonal lab reagents for research
  • Compare Pcr lab reagents for research

RSS Trends in Genetics

  • Epigenetic perspectives on wheat speciation, adaptation, and development
  • New dimensions in the molecular genetics of insect chemoreception
  • Molecular circuits for genomic recording of cellular events
  • Moving towards sequencing-based metabolomics
  • Intragenomic mutational heterogeneity: structural and functional insights from gene evolution
  • Genomic newborn screening: exploring opportunities and navigating pitfalls while ensuring inclusivity
  • Can genomic analysis actually estimate past population size?
  • Polyploidy in potatoes: challenges and possibilities for climate resilience
  • Genetic databases in the era of ‘DSI’ benefit-sharing
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Categories

  • 4 Host
  • 96T
  • apoptosis
  • array
  • Assay
  • Bacteria
  • DNA
  • Establishment and characterization of HBV-associated B lymphocytes with an immortalization potential.
  • GMO
  • Goat
  • Green
  • Guinea
  • Malignant transformation in a Breast Adenomyoepithelioma Caused by Amplification of c-MYC: A Common pathway to Cancer in a Rare Entity.
  • Mechanisms underlying the activation of TERT transcription and telomerase activity in human cancer: old actors and new players.
  • Pig
  • Pigeon
  • PITX1 protein interacts with ZCCHC10 to regulate hTERT mRNA transcription.
  • Plant
  • plasmid
  • Plate
  • PTPN14 degradation by high-risk human papillomavirus E7 limits keratinocyte differentiation and contributes to HPV-mediated oncogenesis.
  • RNA
  • SMR peptide antagonizes mortalin promoted release of extracellular vesicles and affects mortalin protection from complement-dependent cytotoxicity in breast cancer cells and leukemia cells.

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  • NCBI References of Lab polyclonals
  • Suppliers of Lab recombinants
  • Distributors of Lab monoclonals
  • Compare monoclonal lab reagents for research
  • Compare Pcr lab reagents for research
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